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(5) This process disrupted vascular homeostasis involving angiopoietins (Ang1/2) and their tyrosine kinase receptors (Tie1 and Tie2) axis dysregulation

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ROS-related anticancer mechanisms include mitochondrial dysfunction, p53-dependent apoptosis, iron-dependent ferroptosis, activation of endoplasmic reticulum stress, inhibition of growth signaling pathways (such as the epidermal growth factor pathway, EGF), among others

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