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There appears to be an anomalous storage of mitochondrial ATP synthase subunit c that is neither the result of mutation nor enhanced expression of the protein but rather a slower degradation of the mitochondrial ATP synthase in comparison with normal cells [116]

AIFM1 mutation presenting with fatal encephalomyopathy and mitochondrial disease in an infant

St Germain DL, Galton VA

Park J-M, Lee D-H, Kim D-H (2023) Redefining the role of AMPK in autophagy and the energy stress response

155 Cellular senescence Cellular senescence is a self-defense mechanism triggered by internal and external stimulation, playing a pivotal role in organismal development and post-injury repair

Considering the possible pathomechanisms of the observed AEs, the authors once again postulated that they were mainly related to caffeine action