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The development of acute pancreatitis in all periods of the study resulted in a deficiency of reduced glutathione in the pancreas, small intestine mucosa, liver, and blood, which is attributed to the neutralization of lipoperoxides and active forms of oxygen

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The redox potential shifted from around 228 mV (WT) to 233 mV, counterintuitively a more reducing state, in the absence of GSH (Fig