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Folic acid supplementation prevents high fructose-induced non-alcoholic fatty liver disease by activating the Ampk and Lkb1 signaling pathways

These findings suggest a potential mechanism whereby SIRT1-mediated promoter deacetylation may contribute to the transcriptional repression of PGC-1 under conditions of hemodynamic stress

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[DOI] [PubMed] [Google Scholar] 90.Mukherji A, Dachraoui M, Baumert TF

Discontinuation of HMA Therapy after Achieving Complete or Partial Response: Retrospective Analysis of Survival after Long-Term Follow up