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Evidence that serum calcium oxalate supersaturation is a consequence of oxalate retention in patients with chronic renal failure

CDAHFD protects against conditional hepatocyte-specific Gpx4 deficiency-induced death As a reverse strategy, we explored the effect of depleting Gpx4 in CDAHFD-induced MASH through tamoxifen-inducible hepatocyte-specific Gpx4 deficiency

Notably, HCQ treatment reversed LPS-induced upregulation of MDA and cytoplasmic ROS and downregulation of GSH and GPx in monocytes, which had some implications for ferroptosis (80)