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In contrast to adipose tissue, capsaicin engages a complementary thermogenic mechanism in skeletal muscle that is largely independent of both UCP1 and direct TRPV1 activation

Previous studies have shown that melatonin decreased the formation of 8-hydroxy-2-deoxyguanosine, a DNA damage product resulting from free radicals, 60 to 70 times more efficiently than certain traditional antioxidants (Shirazi et al., 2012)

This similarity suggests that the altered energy metabolism at baseline observed in ME/CFS may compromise the ability of these patients to respond to exercise in the same way as healthy individuals

It contributes to: Cellular antioxidant defense Redox signaling and enzyme regulation Normal metabolism of certain drugs and environmental compounds Recycling of other antioxidant systems Immune-cell function Mitochondrial protection Protein structure and cellular repair Glutathione is not a magical cleanse

AMPK activation

[PubMed] [EuropePMC] McGill MR, Jaeschke H