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How to accurately kill cancer cells and prevent the tumor from continuing to grow has always been a key issue in cancer treatment

Although these reactions are evidently too slow to function as a stress response, they have been recognized to be a molecular clock of ageing 52,53

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We also identified mutations in antimalarial resistance orthologs, as well as balancing selection in 13 of the potential blood-stage target genes, which include apical membrane antigen 1 ( Pvama1 ), Pvdbp , erythrocyte binding protein ( Pvebp ), merozoite surface protein 1 ( Pvmsp1 ), five Pvrbps , and three tryptophan-rich antigens ( Pvtrags )