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doi:10.5665/sleep.3298

Early-onset severe neuromuscular phenotype associated with compound heterozygosity for opa1 mutations

Across Achilles tendon detachment [1] , medial collateral ligament transection [4] , myotendinous junction separation [14] , gastric ulcer models [5] , colocutaneous fistula models [6] , spinal cord compression injury [10] , and several CNS perturbation paradigms [11] , administered BPC-157 consistently outperformed untreated controls on the relevant outcome measures

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However, its concentration drops significantly with age, by more than half after the age of 60 [2]

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