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Autophagic degradation of nuclear components in mammalian cells

This Research Topic is dedicated to presenting recent advances in the understanding of GI-tract manifestations and the central and peripheral molecular regulators of new mechanisms of GI ulceration and healing including, for instance, mucosal repair which involves cellular restitution and lasts seconds and minutes as well as the organ healing involving the proliferation and maturation phases lasting hours and days

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Ribonucleotide reductase-mediated increase in dATP improves cardiac performance via myosin activation in a large animal model of heart failure

Accordingly, Tgfbr2 loss in hepatocytes ameliorates HFD-induced NAFLD by improving mitochondrial biogenesis