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Barkovich, A

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516 In experimental autoimmune encephalomyelitis (EAE) mice, CNS expression of IFN before disease onset prevents oligodendrocyte death, demyelination and axonal degeneration

Clinical reports document taste alterations and xerostomia in patients using semaglutide 47 , with studies implicating GLP-1R expression in mammalian taste buds and their role in modulating gustatory sensitivity 48

1B), providing strong evidence of the growth arrest

In particular, riboflavin deficiency in rats resulted in reduced levels of myelin lipids, cerebrosides, sphingomyelin, and phosphatidylethanolamine in the cerebrum and cerebellum and, consequently, led to the impairment of brain development and maturation [20]