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S., Pereira, K

Hashemi Tari, S., Sohouli, M

EomDWLeeJHKimYJHwangGSKimSNKwakJHet al

PC is the fundamental backbone of liposome technology, creating a tight, structured bilayer that prevents the glutathione from oxidizing prematurely

Such modifications can result in loss of enzymatic activity, altered cellular signaling, and degradation of structural proteins, contributing to cellular dysfunction and death [36, 37]

Current pharmacological interventions for oxidative stress in the nervous systemsuch as edaravone (15, 16) and N-acetylcysteine (16, 17)act on single targets and employ relatively narrow mechanisms, and are associated with dose-limiting adverse effects and loss-of-benefit phenomena (e.g., end-of-dose deterioration and motor complications), rather than durable disease modification (18)