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doi: 10.3389/fnut.2022.950062 129 ZouCChenYLiHLiWWeiJLiZet al

Hcy homocysteine

The specific reason for this may be related to the ability of TZDs to inhibit the entry of pyruvate into the TCA cycle [167]

Beyond a target for therapeutic intervention, monitoring GSH catabolism holds value as a cancer diagnostic

However, there was a negligible effect on the AGS and HDF normal cell lines

This process subsequently contributes to activating the nuclear factor kappaB (NF-B) pathway, upregulated glutamine transporters such as SLC38A2 and SLC7A5, and ultimately increased glutamine metabolism in cancer cells [128]