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Amyloid oligomer proteins which are formed through fibrilization, accumulate and can selectively damage neurons, is a condition common in numerous neurodegenerative disorders

Yang L, Zheng XL, Sun H, Zhong YJ, Wang Q, He HN, et al

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Mitochondria-targeted prodrug nanoassemblies for efficient ferroptosis-based therapy via devastating ferroptosis defense systems

Additionally, ATF4 and CCAAT/enhancer-binding protein (CEBP) homologous protein (CHOP) mRNA are highly expressed, along with activated PERK pathway in VSMCs and macrophages at this stage (76) (Figure 2A)

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