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Under pathological stimulation, overactivation of microglia leads to the release of large amounts of pro-inflammatory factors, ROS, and nitric oxide (NO), triggering uncontrolled inflammatory responses that ultimately result in neuronal damage and sustained neuroinflammation [68]

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This is perhaps hampered by the difficulty and increased workload in isolating single colonies from population sweeps, the lack of paired biopsy samples from the same patient and the increased sequencing costs such investigations incur

( Junyun Cheng et al., Massively parallel CRISPR-based genetic perturbation screening at single-cell resolution

frequency of infection with this bacterium is associated with several factors such as virulence factors (e.g

Thymosin beta-4 might be involved in the healing of the corneal surface