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Hyperhomocysteinemia and endothelial dysfunction in patients with cerebral vascular and autoimmune diseases

Overall, we aimed to investigate the potential of 3BP, a small molecule metabolic poison, to selectively target TNBC cells by exploiting the overexpression of MCT1 combined with the Warburg phenotype

To further assess the contribution of established Osimertinib targets, including EGFR and MKNK2, we reanalyzed drug sensitivity data across multiple GBM cell lines from the Dependency Map (DepMap) database

L-DOPA-treatment in primates disrupts the expression of A(2A) adenosine-CB(1) cannabinoid-D(2) dopamine receptor heteromers in the caudate nucleus

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