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Although some feel that MxA may be a primary event, classic studies have shown that this is a secondary innate immunity molecule and in distinct contrast to the primary process of complement-mediated microvasculopathy, as highlighted above, which: a) precedes all pathologic events, b) explains the noted ischemia and perifascicular atrophy, and c) explains the impressive improvement after complement inhibition by IVIg based on two controlled trials (34)

10.1023/B:BIBU.0000007716.17865.c8 [DOI] [PubMed] [Google Scholar] Vogg M