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Unexpectedly, the T H 17 group exhibits a higher level of GAD than the iT reg group and was the only group that expressed a high level of the GAB-catabolizing enzyme 4-aminobutyrate aminotransferase (ABAT), indicating increased GAB catabolism in T H 17 cells but not in iT reg cells (Fig

AMPK activation triggers PGC-1 expression, enhancing mitochondrial biogenesis and OXPHOS capacity [24]

Subsequently, this leads to oxidative membrane damage, rupture, and ferroptotic cell death