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This exacerbates lipid peroxidation, drives excessive ROS accumulation and neuroinflammation, and ultimately triggers ferroptosis, resulting in irreversible neuronal damage and neurotoxicity (Table 1), suggesting that therapeutic targeting of ferroptosis may offer a promising strategy for the prevention and treatment of NDDs

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Mild hyperhomocysteinemia was not associated with higher rates of stroke

Additionally, development of novel NAD+ precursors and delivery methods aims to improve bioavailability and tissue-specific targeting

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Selenium, cadmium (Cd), arsenic (As), and lead (Pb) in particular have attracted a lot of study due to their complex relationships with thyroid function and trace elements [3]