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Activation of GABA A receptors on alpha cells leads to hyperpolarization and suppression of glucagon and glutamate secretion [103, 111]

Pathogenic alpha-synuclein aggregates preferentially bind to mitochondria and affect cellular respiration

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Integrative analysis of gut microbiome and host transcriptome reveal novel molecular signatures in Hashimotos thyroiditis

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However, these models are not amenable to high-throughput treatment screens