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Conversely, if Drug A is an inhibitor of a CYP that also metabolizes Drug B, it will slow metabolism of Drug B, possibly raising its concentration to toxic levels

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The accumulation of ROS can initiate apoptosis through several key mechanisms: Firstly, ROS can damage the mitochondrial membrane, leading to a decrease in mitochondrial membrane potential, which in turn promotes the release of proteins like cytochrome C (Liu et al., 2024c)

Reduction of TMAO level enhances the stability of carotid atherosclerotic plaque through promoting macrophage M2 polarization and efferocytosis

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Martinez and Tach, 2001)