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J Biol Chem 285(22):1663216642

GCLC genetic variation shapes glutathione synthetic capacity through two primary mechanisms: The GAG trinucleotide repeat polymorphism

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Furthermore, they showed that the inhibition of DNA methylation using decitabine resulted in the upregulation of ADH1B and ADH1C , whereas the inhibition of histone deacetylases (HDACs) using trigonelline A promoted ADH1C expression

Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis

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