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The cell initiates apoptotic death when it senses that its environment of physical state has been vigorously compromised

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This vulnerability is compounded by an age-dependent decline in endogenous antioxidant defenses, contributing significantly to neuronal dysfunction and neurodegenerative pathogenesis

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While cytosol, peroxisomes, mitochondria, and plastids in Arabidopsis wild-type plants maintain a highly reduced glutathione buffer with redox potentials of less than 310 mV, the ER is far less reducing with E GSH values of less than 240 mV (Meyer et al., E GSH with high specificity due to selective mediation of thiol/disulfide exchange reaction between glutathione and roGFP by GRXs (Meyer et al., E GSH is outside the measuring range of these probes thus limiting their usability