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Thus, the transition from physiological to pathological fibrosis is driven by impaired apoptosis of myofibroblasts, as well as excessive ECM deposition and accumulation, resembling a persistent wound-healing response

Yuejun Zheng, et al

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COX-2, cyclo-oxygenase-2

Unraveling the molecular mechanisms of cell migration impairment and apoptosis associated with steroid sulfatase deficiency: implications for X-linked ichthyosis

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