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Stokes CS, Gluud LL, Casper M, Lammert F

Nov 2012;87(11):1017-1021

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This effect was not restricted to Venetoclax, as knockout or pharmacologic inhibition of ABCC1 also sensitized AML cells to other BH3 mimetics, including ABT-737, Navitoclax and the novel BCL-2/BCL-xL inhibitor AZD-4320

This TFAP2B-expressing NCSC state emerges in metabolically active ALDH1A3-high cells through acetaldehyde metabolism, creating an acetyl donor for histone H3 modification to promote neural crest lineage gene expression 69 (Fig