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Protein S-glutathionylation (PSSG) may modify the activity of a large number of cell proteins, including metabolic, structural, cytoskeletal, and signaling proteins

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demonstrated that polymorphism of the optic atrophy 1 (OPA1) gene, which encodes a dynamin-related mitochondrial protein, was related to the pathology of OAG.[43] Additionally, we previously showed that mitochondrial DNA damage in blood cells, represented by an increased mitochondrial/nuclear DNA (mtDNA/nDNA) ratio, was associated with abnormal BF (MBR) in the ONH in glaucoma patients.[44] Taking these past results together with those of this study, it is thus reasonable to consider that systemic mitochondrial dysfunction influences hemodynamics in the ONH

This review comprehensively explores the mechanism of action of oxidative stress, mitochondrial apoptosis, autophagy dysfunction, calcium imbalance and ferroptosis on DFU pathogenesis

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112 NishinoI.SpinazzolaA.PapadimitriouA.HammansS.SteinerI.HahnC