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With age, your stomach cells become less efficient and secrete less hydrochloric acid, which means you absorb less B12

These changes occurred through direct effects on adipocyte biology rather than appetite suppression, as research models maintained normal feeding behavior while exhibiting significant reductions in individual adipocyte size

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In preclinical mouse models, this produced significant reductions in body weight and fat mass without changes in food intake

If the safety and modest efficacy data from the published trials translate to our patient population, it would give us a tool that acts directly on fat cell metabolism rather than appetite, with a side effect profile that appears extremely well-tolerated