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This heightens susceptibility to the toxic effects of iron overload, disrupting intestinal mucosal homeostasis and triggering local inflammation and cell death, ultimately precipitating the onset of gut-associated diseases ( Iron overload-related diseases rank among the most prevalent genetic disorders in humans, characterized pathologically by systemic iron accumulation resulting from excessive dietary iron absorption and iron-induced oxidative stress responses ( 3 Ferroptosis and inflammatory bowel disease The imbalance in ferroptosis regulation plays a key role in the pathogenesis of various diseases

The influence of physiological and pathological perturbations on bloodbrain barrier function

10.1016/j.tube.2012.07.003 15 GaucherC.BoudierA.BonettiJ.ClarotI.LeroyP.ParentM

41 ), we identified a key role of antioxidant systems that rely on reducing equivalents such as GSH and NADPH to cope with oxidant stress such as G6PD deficiency

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TABLE 1 Overview of the resistance mechanisms of fosfomycin