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J Orthop Sports Phys Ther

c-myc protooncogene polypeptide expression in endometriosis

Lipoate and OGDHc-E2 levels were measured by immunoblot

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APAP overdose leads to excessive generation of the highly reactive metabolite n-acetyl-p-benzoquinone imine (NAPQI), which depletes glutathione (GSH), increases oxidative stress, and causes mitochondrial dysfunction, finally triggering immune response-mediated hepatotoxicity and massive hepatocytes necrosis [15, 16]

The UCP2/SIRT3/PGC-1 signaling axis, activated by matrine, plays an important role in limiting oxidative damage by decreasing mtROS production, inhibiting ferroptosis, and preserving mitochondrial function while also implicating peroxisomal metabolism in redox homeostasis (77)