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Axonal damage, evaluated by the presence of amyloid precursor protein (APP + ) deposits, firstly appears at remission (Fig

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This hypercatabolism is evident by our data demonstrating depletion of amino acids and upregulation of the urea cycle as judged by elevated levels of urea cycle metabolites such as citrulline

doi:10.1016/j.ejca.2010.11.003

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