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Platelet-derived growth factor receptor- induces contraction knots and inflammatory pain-like behavior in a rat model of myofascial trigger points

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Mice with targeted disruptions of SUR1 [7, 8] or the inward rectifying K + channel [9, 10] were still capable of secreting insulin in response to glucose stimulation, suggesting that GSIS does not solely operate through the K ATP channel

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Additionally, the axon count in the ES groups closely resembled that of the autograft group, with enhanced myelin thickness observed in animals treated with conductive conduits and ES compared to other groups