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In support of the NRF2 pathway being a potential target for the treatment of ALS, studies have shown that NRF2 overexpression in astrocytes provided neuroprotection against mutated SOD1-induced toxicity and increased the median survival of mutant SOD1 transgenic mice (Vargas et al., 2008

l-Cysteine suppresses hypoxia-ischemia injury in neonatal mice by reducing glial activation, promoting autophagic flux and mediating synaptic modification via H(2)S formation

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doi: 10.1097/MED.0b013e32833363cc 185 Rog-ZielinskaEAThomsonAKenyonCJBrownsteinDGMoranCMSzumskaDet al