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By focusing on a subset of aged degus that show AD-like behavioral deficits and correlative neuropathology, we establish outbred degus as a natural model of sporadic AD and demonstrate the potential importance of wild-type outbred genetic backgrounds for AD pathogenesis. This study was motivated by the need to settle earlier debates of whether degus can be a useful natural model of AD

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More research is required in humans although there is some evidence supporting the use of acetyl-L-carnitine to treat mild cognitive impairment and mild Alzheimers disease

Accelerated aging phenotype in mice with conditional deficiency for mitochondrial superoxide dismutase in the connective tissue

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