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Continued research can position Rg1 as a valuable therapeutic option for PD, offering improved therapeutic solutions for patients

Loss of cGAS abolished senescence phenotypes and senescence-associated -galactosidase (SA--Gal) activity, suggesting that cGAS is essential for cellular senescence (Yang et al., 2017)

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Gastroenterology 135(4):10471051 Hanawa N et al (2008) Role of JNK translocation to mitochondria leading to inhibition of mitochondria bioenergetics in acetaminophen-induced liver injury