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Proline metabolism has been confirmed to enhance the tumorigenesis in liver cancer as two enzymes corresponding to proline biosynthesis are upregulated (pyrroline-5-carboxylate reductase (PYCR1), aldehyde dehydrogenase 18 family member A1 (ALDH18A1)), and one proline catabolic enzyme is downregulated (proline dehydrogenase (PRODH)) ( Moreover, the interplays between metabolism and tumor microenvironment play crucial roles in the cancerous liver

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Table 1 summarizes the physiological and therapeutic activities of sea buckthorn preparations through in vivo and in vitro experimentations on several experimental models

Few studies have also developed an LS model for neuroprotection studies by using the complex I inhibitor MPTP (1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine)