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Mutations in both Y196 and Y247 gave rise to a mutant that had lost the ability to self-AMPylate but retained the activity to convert PR-Ub into ADPR-Ub (Fig

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Additional cellular experiments indicate that podocyte-specific NLRP3 inflammasome activation may promote aseptic inflammation and glomerular injury in DKD kidney tissue (200)

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[66] [67] Nine inherited neurodegenerative diseases are caused by the expansion of the CAG trinucleotide and polyQ tract, including Huntington's disease and the spinocerebellar ataxias