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Administration of exogenous carnitine as an adjunct to VPA therapy is thought to restore beta-oxidation to mitochondria cells of the liver, accept toxic acyl moieties from CoA, and relieve the inhibition of urea synthesis.[3] However, our findings suggested that the effective utilization of levocarnitine transport into mitochondria, which may be individually different, might play an important role to lead these favorable processes

Neither high (Figure 5, group 2) nor low levels (Figure 5, groups 5 and 7) of plasma medium and long chain acylcarnitines noted in our work correlate with described genetic disorders of mitochondrial fatty acid or organic acid metabolism in humans (64)

doi: 10.1016/0002-8703(91)90861-b 4 AnwarS.BhandariU.PandaB

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Mitochondria, cybrids, aging, and Alzheimers disease