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Furthermore, 2-DG-mediated inhibition of glycolysis effectively reverses various electrophysiological alterations induced by epilepsy, suggesting that targeting lactate metabolism pathways could be a promising approach for the development of novel antiepileptic drugs [195] (Supplemental Table 2)

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The distinction carries several practical consequences that the literature illustrates: One important corollary: because MK-677 depends on a functioning pituitary, its effect is fundamentally capped by the somatotrophs capacity to respond