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3p), indicating that the TCA cycle is also functionally impaired

Results suggest that the antiproliferative effects of NAC and Mito 10 -NAC are unlikely to be related to their antioxidant mechanism (i.e., scavenging of reactive oxygen species) or to the sulfhydryl group-dependent redox modulatory effects

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Oxidative stress might result the activation of the proinflammatory transcription factor, NF-B, impaired antiprotease defences, cellular senescence, and corticosteroid resistance