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The continued development of LRRK2 inhibitors and biomimetic nanotherapies highlights the translational potential of mechanism-based interventions

Moreover, Glu transporter expression in the cerebral cortex of AD patients and VGLUT1 significantly decreased, possibly resulting in poor clearance of glutamic acid in the synaptic cleft, leading to excitatory toxicity

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Qki regulates myelinogenesis through Srebp2-dependent cholesterol biosynthesis

These collective gaps point to clear directions for future research

GO enrichment analysis revealed significant enrichment of differentially expressed genes (DEGs) involved in cell apoptosis and mitochondrial dysfunction process (Fig