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10.1371/journal.pone.0026753 118

These neuroplastic changes are thought to be mediated by changes in glutamate receptor sensitivity [72, 73] which modulate dopamine firing in the ventral tegmental area and the nucleus accumbens region [62]

Several studies have demonstrated its ability to inhibit NF-B activation, a key factor in promoting carcinogen-induced processes such as apoptosis suppression, cellular transformation, invasion, metastasis, chemoresistance, and inflammation (522, 523)