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In the setting of hyperlipidemia, the pathological effect of transsulfuration pathway on redox status is not that simple, because in normal physiological settings, the flux of homocysteine to GSH through the transsulfuration pathway can be compensatively enhanced in response to the oxidative stress in order to maintain the oxidant/antioxidant balance whereas antioxidant treatment elicit the opposite effect on transsulfuration pathway [8]

Comparing adiposity profiles in three mouse models with altered GH signaling

Although protective drugs have also been discovered, the studies had limitations, including their small sample sizes and failure to explore the mechanisms in-depth, and the clinical relevance of the findings has yet to be verified

Product-quality risk is unquantified because grey-market vials are not independently characterized

Curcumin-loaded porphyrin micelles (T-MP) were successfully prepared with a size smaller than 50 nm using a surfactant-stripping method