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Thus, the upregulation of PRMT2 activates ferroptosis via the -catenin/GPX4 axis and participates in lipopolysaccharide-induced depressive behavior

Furthermore, HMGCS2 overexpression stimulated ketogenesis while its silencing inhibited APP degradation, which was re-established by acetoacetate

The immunomodulatory effects of these compounds are not restricted to cytokine modulation but also extend to cellular interactions within the immune system