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In glycolysis, ROS oxidize GAPDH and PKM2, altering their activity, thereby inhibiting glucose uptake, while simultaneously enhancing glucose uptake and metabolism through HIF1 stabilization

Lipoic acid as a novel treatment for Alzheimers disease and related demenias

Targeting antioxidant pathways may therefore offer a novel avenue for CRC treatment in the future

However, when present in excess, it contributes to the development of fatty deposits in the arteries, which can restrict blood flow and lead to serious health issues such as heart attacks and ischemic strokes [1][2]

FAO not only supports ATP production but also promotes tumor progression, metastasis, and therapeutic resistance by regulating ROS levels, lipid metabolism-related transcription factors such as peroxisome proliferator-activated receptors (PPARs), and components of the AMP-activated protein kinase (AMPK) signaling axis [15, 16]