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Biomarkers that could identify metabolic disturbances and NAFLD early, as well as supportive therapies, could be beneficial, and to that end I reviewed two current articles: Mardinoglu, M., et

Neu5Ac facilitates SLC3A2 binding to ubiquitin, initiating P62-mediated degradation, which leads to LPO and ferroptosis in high-fat-fed ApoE / mice

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