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This shifting ratio indicates that some factor was introduced into the population that could induce ASD even after brain development had proceeded for years on a relatively normal trajectory.A second distinct temporal relationship (Fig

Figure 3 illustrates the interaction between peripheral inflammation, brain inflammation, and the HPA axis, highlighting their roles in stress-related disorders

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Apoptosis and inflammation associated gene expressions in monocrotaline-induced pulmonary hypertensive rats after bosentan treatment