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One potential mechanism that could account for estrogen effects on the intestine is through its alterations in TJ and adhesion molecules which would alter intestinal permeability [94]

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Crohns disease pathobiont adherent-invasive E coli disrupts epithelial mitochondrial networks with implications for gut permeability

AMP-activated protein kinase alpha2 contributes to acute and chronic hyperuricemic nephropathy via renal urate deposition in a mouse model

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