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Although this increase in systemic exposure to the metabolites seems counterintuitive with respect to CYP3A inhibition, the inhibition of their secondary metabolism via UGT2B4/2B7 by kratom is a potential explanation

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doi: 10.3390/biomedicines10061350 192 ZhangYHuangPLiuXXiangYZhangTWuYet al

Similar content being viewed by others 1 Introduction Hashimotos thyroiditis (HT) is an autoimmune disorder of the thyroid with a multifactorial etiology involving both genetic predisposition and environmental triggers