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Receptor for advanced glycation end products (RAGE) is a key factor for plaque destabilization in diabetes mellitus, where its downregulation may suppress atherosclerotic plaque development, an effect mediated by NF-B inhibition (100, 101)

Psychopharmacology, 238 , 441-451

Redox Biol 81:103548

Bronchodilator mechanisms for beta-2 agonists and theophylline

Suppression of NF-kappaB activation by PDLIM2 restrains hepatic lipogenesis and inflammation in high fat diet induced mice

Collectively, these findings indicate that AI can optimize the diagnostic precision and facilitate personalized treatment recommendations, enabling healthcare professionals to optimize cardiovascular disease management, minimize unnecessary diagnostic procedures and interventions, and ultimately improve patient outcomes and quality of life