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Guo et al.[111] and Martin-Sanchez et al.[112] confirmed that iron overload and lipid peroxidation existed in an FA-induced AKI (FA-AKI) mouse model, and ferroptosis inhibition could effectively improve renal function and ameliorate kidney damage in FA-AKI

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The GSH concentration was not affected in the mucosa at 5% SI and 75% SI and in erythrocytes by adding dietary GSH both on d5 and d14