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The impaired adipogenesis is associated with stress of the endoplasmic reticulum (ER) and elevated expression of unfolded protein response (UPR), both can exacerbate the proinflammatory phenotype of preadipocytes and adipocytes [58]

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In agreement with these data, we have observed that UDCA alone improves the intestinal Ca 2+ absorption by increasing the amount of Ca 2+ transporters involved in the transcellular Ca 2+ pathway via activation of the VDR gene and protein expression

The additive effect of fasting in this situation may be due to necessary downregulation of other compensatory lipolytic inhibitors or compositional changes allowing for an increased activity or localization of ATGL that remain to be determined